The interplay between DNA repair and autophagy in cancer therapy


First Post

Below is another example of how establishment science doesn’t understand the basics physics of life. The lung cancer tissue samples are sugar burners. They are metabolically imbalanced. They generate an excess of free radicals which if not controlled would lead to apoptosis. They are trying to survive by balancing the dangerous but efficient Free radical producing electron transport system with safer aerobic glycolysis. 

When these efforts are insufficient CB2 activity is used to turn on fat burning so of course the cell will survive, and the tumors will grow bigger, because they’re not busy dying. CB2 activity turns on fat burning as their own data shows with the Down regulation of AKT etc.


The oncogenic role of CB2 in the progression of non-small-cell lung cancer. - PubMed - NCBI

Biomed Pharmacother. 2019 Jun 5;117:109080. doi: 10.1016/j.biopha.2019.109080. [Epub ahead…his is your first blog entry. What are you waiting for? Replace this text with some of your own and start blogging!

The interplay between DNA repair and autophagy in cancer therapy

The paper below clearly shows that sugar promotes radiation resistance in cancer cells by enhancing certain DNA repair capacities in response to DNA damage.  When the say glucose stimulates glycolysis, what they have shon is that it stimulates aerobic glycolysis (Warburg effect), one of the main pathways that cancer cells used to divert from the electron transport system in order to reduce apoptosis drbob

© drbob 2018